Adjuvant Endocrine Therapy in Postmenopausal Women
with Breast Cancer
Background Information
Approximately three
quarters of all invasive breast tumours are oestrogen or
progesterone receptor positive, including at least half of all breast cancers
in premenopausal women. This means
that the cancer cells express receptors for these hormones on their surface.1
Adjuvant endocrine therapy describes the treatment of these cancers with
additional hormone drugs, after surgery to remove the tumour, in order to help
stop the disease coming back and increase the chance of cure.
Adjuvant therapy may
include chemotherapy, radiation therapy or endocrine (hormone) therapy. There
are different types of adjuvant endocrine therapy, all of which work in
slightly different ways to prevent breast cancer recurrence.2
Adjuvant endocrine
therapy is usually given after surgery and radiotherapy for breast cancer, where
intervention with hormone therapy has been proven to reduce the risk of
recurrence.2,3 Women with breast cancer may also be treated with adjuvant
endocrine therapy prior, or subsequent to, chemotherapy.2 In
general, women remain on adjuvant endocrine therapy for five years, however,
new evidence has emerged showing the value of extending the treatment duration.4
Adjuvant endocrine
therapy is recommended for all women with tumours that have been assessed as
responsive to hormone treatment, also known as Ôhormone-responsiveÕ breast cancer.4
To determine if the breast cancer will respond to hormones, an
oestrogen/progesterone receptor test is carried out on the tumour.5 If
positive, the patient will normally be prescribed adjuvant endocrine therapy
after their operation.5
Hormones are secretory substances that control the
growth and activity of normal cells.3 Although they do not usually
affect cancer cells, in breast cancer, the situation is different and both the female
hormones, oestrogen and progesterone, have been shown to have a
growth-promoting impact on certain breast cancer cells.3 Depending
on the particular type of hormone treatment used, adjuvant endocrine therapy
exerts its effects either through the blockade of oestrogen production or by
preventing oestrogen from reaching its target receptors on cancer cells.6
Tamoxifen is the oldest and most commonly used adjuvant
endocrine therapy.3,7,8 It is an anti-oestrogen drug which works by
preventing the hormone from attaching to breast cancer cells and encouraging
them to grow.8 Tamoxifen can reduce the risk of recurrence by about
30% and, on average, has been shown to decrease the risk of developing a second
breast cancer by around 50%.8 It is taken once daily in tablet form,
usually for a period of five years.7 Common side effects associated
with tamoxifen include menopausal symptoms, such as hot flushes and weight gain. Tamoxifen is also associated with an
increased risk of thrombosis and endometrial cancer.3,8
Aromatase inhibitors, the next generation of endocrine
therapy, block the small amount of oestrogen that is produced by the adrenal
glands which lie just above the kidneys.9 They work by inhibiting
the activity of the enzyme, aromatase, which is needed to convert androgen
hormones into oestrogen.9 These drugs are used in post-menopausal
women whose ovaries have shut down and where the only remaining source of
oestrogen production is via the adrenal route.9 Aromatase inhibitors
are used to treat post-menopausal women with early breast cancer in order to
reduce risk of recurrence and there is growing evidence to demonstrate the
superiority of these agents in comparison to tamoxifen. 3,10,11 Aromatase inhibitors may be used as an
alternative or subsequent therapy to tamoxifen. There is evidence that risk of recurrence can be reduced
further by switching to exemestane or anastrozole after two to three years
of tamoxifen.3,
12, 13 Risk of recurrence can be further reduced by treatment with letrozole
after five years of tamoxifen.3, 4 Aromatase inhibitors are taken
once daily for a period of several months or years.9 Like tamoxifen,
side effects include menopausal symptoms such as hot flushes, vaginal dryness
and tiredness however these agents are not associated with the side effects of
thrombosis and endometrial cancer.9 Other potential adverse effects
include increased osteopenia, nausea, headaches, diarrhea and muscle pain.9
Many studies have highlighted that adjuvant endocrine
therapy is an essential tool in the fight against breast cancer as it decreases
the risk of recurrence by at least one third.7 Analysis of almost
37,000 women with early breast cancer showed that adjuvant tamoxifen treatment (anti-oestrogen
therapy) substantially improved the 10-year survival of women with ER-positive
tumours and those whose tumours were of unknown ER status.14 Similarly,
another analysis of recurrence among ER+ patientsÕ found that use of tamoxifen
therapy significantly improved patients outcomes.15 However, even
with adjuvant endocrine therapy, more than 20% of node-negative patients had
their disease recur within 15 years after diagnosis.15
A number of studies have compared tamoxifen with
aromatase inhibitors in the early breast cancer setting and have shown that there
is significant evidence to support the use of aromatase inhibitors over
tamoxifen.10, 11, 16 For example a trial10 incorporating
both a head-to-head comparison of letrozole with tamoxifen and a sequencing of
both agents to determine the most effective approach to minimise the risk of
breast cancer recurrence showed that letrozole:
For this reason a number of recent guidelines -
including those from St Gallen and the National Comprehensive Cancer Network
– have recommended that aromatase inhibitors be used as part of the
optimal adjuvant treatment of postmenopausal women with hormone-sensitive
breast cancer.17, 18
Despite the fact that adjuvant endocrine therapy can
reduce recurrence after diagnosis and initial treatment, there is an ongoing
risk of recurrence.7 A study that evaluated the risk of breast
cancer recurrence following adjuvant endocrine therapy in over 2,400 women with
early breast cancer revealed a substantial and continuing risk of recurrence
long after completion of five years of standard adjuvant endocrine therapy.10
Another study showed that more than half of all breast cancer recurrences and
two-thirds of all breast cancer deaths occur after 5 years of standard
tamoxifen therapy.4 Despite the residual risk of recurrence after 5
years of adjuvant endocrine therapy, continued use of tamoxifen after this
period has been found to be associated with an increase in serious adverse
events without further efficacy benefits.15
Therefore, a number of studies have looked at the
benefit of using AIs in the extended adjuvant setting. For example a trial4 looking
at the effectiveness of letrozole vs. placebo in women who have received prior
standard adjuvant tamoxifen therapy for five years, showed that letrozole:
Significantly, the interim data from the study led the
investigators to unblind (open the code of the treatment taken) the trial early
so that those patients taking placebo could be offered the opportunity to
switch to letrozole. A further
analysis of the data showed that the women (n=1655) who opted to take letrozole
following a prolonged delay after completing tamoxifen experienced a
significant improvement in outcome (disease-free survival, distant disease-free
survival, and overall survival) in comparison to those who opted to take no
further treatment (n=613).19
References
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